Chapter 15 Eyes

External Anatomy of the Eye

The eye is housed in a bony orbital cavity, which is cushioned by a protective layer of fat. This structure safeguards the eye from trauma and provides stability.

  • Palpebral fissure: The elliptical open space between the upper and lower eyelids.
  • Lower lid margin: Located at the limbus, where the cornea and sclera meet.
  • Canthus: The corner of the eye where the upper and lower lids meet. The inner canthus contains the caruncle, a small fleshy mass with sebaceous glands.
  • Tarsal plates: Dense connective tissue structures inside the eyelids containing the meibomian glands. These modified sebaceous glands secrete an oily substance that prevents evaporation of tears and keeps the eyelids lubricated.
  • Conjunctiva: A transparent mucous membrane that covers the sclera (bulbar conjunctiva) and lines the inside of the eyelids (palpebral conjunctiva).
  • Cornea: A transparent layer that covers the iris and pupil, serving as part of the eye’s refracting system.
  • Lacrimal apparatus: Produces and drains tears, providing constant irrigation to keep the eye moist and free of debris.
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Extraocular Muscles

Six extraocular muscles attach the eyeball to the orbit. Their coordinated movements ensure binocular vision by maintaining parallel alignment of both eyes.

  • Four rectus muscles: Superior, inferior, lateral, and medial rectus muscles.
  • Two oblique muscles: Superior and inferior oblique muscles.
  • Movement types: These muscles allow both straight and rotary eye movements.
  • Conjugate movement: When both eyes move in a coordinated way, keeping visual axes parallel. This allows the brain to process a single, fused image.

Cranial Nerve Innervation of Extraocular Muscles

The extraocular muscles are controlled by three cranial nerves: Note: Abducens means Abduction. Trochlear go to superior Oblique. Rest III.

Cranial Nerve
Name
Innervated Muscle(s)
Action
CN VI
Abducens
Lateral rectus
Abducts the eye (outward)
CN IV
Trochlear
Superior oblique
Rotates eye downward and inward
CN III
Oculomotor
Superior rectus, Inferior rectus, Medial rectus, Inferior oblique
All other movements, eyelid elevation, pupillary constriction

This division of control ensures smooth, coordinated movement in all directions.

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Note: Superior Oblique go down inwards, Inferior oblique goes up inwards, it’s the Opposite.

👉 Key Points

  • The eye is protected by orbital fat, eyelids, and specialized glands.
  • The conjunctiva and cornea act as protective, transparent coverings.
  • Six extraocular muscles provide movement, controlled by CN III, IV, and VI.
  • Proper coordination (conjugate movement) is crucial for binocular vision.

📝 Detail Information

Damage to any of the cranial nerves affecting eye muscles can result in diplopia (double vision) or strabismus (misalignment). For example, CN VI palsy prevents lateral eye movement, leading to inward deviation of the eye. The lacrimal system not only lubricates but also contains enzymes such as lysozyme, which help defend against bacterial infections. The cornea is avascular, relying on tears and aqueous humor for oxygenation and nutrients, making it highly vulnerable to damage yet capable of rapid healing.

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Internal Anatomy of the Eye

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Outer Layer

The outer layer of the eye provides structure and protection:

  • Sclera: This is the tough, fibrous, and protective white covering of the eyeball. It maintains the shape of the eye and serves as the site of attachment for extraocular muscles. Anteriorly, the sclera continues seamlessly with the transparent cornea.
  • Cornea: The cornea is the smooth, transparent dome that covers the iris and pupil. It is a crucial part of the refractive media of the eye, bending incoming light rays so they can be focused on the inner retina.
  • Corneal reflex: Contact with the cornea, even a light touch with a wisp of cotton, produces an involuntary blink in both eyes. This reflex involves:
    • Cranial nerve V (Trigeminal nerve): Carries the afferent (sensory) signal into the brain.
    • Cranial nerve VII (Facial nerve): Sends the efferent (motor) signal to close the eyelids.

Middle Layer

The middle vascular layer contains structures critical for nourishment and light regulation:

  • Choroid: This darkly pigmented layer prevents light from reflecting internally, ensuring clear images. It is also highly vascular, supplying blood to the retina.
  • Iris: The iris functions as a diaphragm, regulating the size of the pupil at its center. Its muscle fibers respond to lighting conditions and visual needs:
    • In bright light or during near vision, the circular muscles contract to constrict the pupil.
    • In dim light or during far vision, the radial muscles contract to dilate the pupil.
  • Pupil: The pupil is the round, central opening of the iris. Its size is determined by the balance between sympathetic and parasympathetic inputs of the autonomic nervous system.
    • Parasympathetic stimulation (CN III, Oculomotor nerve) → constricts the pupil.
    • Sympathetic stimulation → dilates the pupil and elevates the upper eyelid.
  • Pupil size also varies in response to ambient light levels and during accommodation (focusing on near objects).
  • Lens: A transparent, biconvex disc positioned directly behind the pupil. Its main role is to act as a refractive medium, adjusting focus so that light falls on the retina.
  • Aqueous humor: Both the anterior and posterior chambers contain this clear, watery fluid, which is continually produced by the ciliary body. Its functions include:
    • Delivering nutrients to surrounding tissues.
    • Removing metabolic waste.
  • Intraocular pressure (IOP): Determined by the balance between aqueous production and outflow resistance. An imbalance may lead to elevated IOP, as seen in glaucoma.

👉 Key Points

  • The sclera is the protective outer coat; the cornea bends light toward the retina.
  • The corneal reflex tests CN V (afferent) and CN VII (efferent).
  • The choroid nourishes the retina and prevents light scatter.
  • The iris regulates pupil size according to light and vision needs.
  • Pupil size is controlled by CN III (parasympathetic) and sympathetic pathways.
  • The lens fine-tunes focus; aqueous humor maintains IOP and nourishes tissues.

📝 Detail Information

The cornea contributes nearly two-thirds of the eye’s total refractive power, making it essential for sharp vision. Corneal damage (trauma, infection, scarring) severely impairs sight. Autonomic control of the pupil is clinically important: a fixed, dilated pupil may indicate brain injury or increased intracranial pressure, while pinpoint pupils may suggest opioid intoxication. The lens becomes less elastic with age, resulting in presbyopia, the inability to focus on near objects. Regulation of aqueous humor outflow is central to glaucoma management, where elevated IOP can damage the optic nerve and progressively impair vision.

Internal Anatomy: Inner Layer

Retina

The retina is the visual receptive layer of the eye. Its role is to receive light waves and convert them into nerve impulses that are transmitted to the brain for interpretation. This transformation allows us to perceive images.

During examination with an ophthalmoscope, several important retinal structures can be visualized:

  • Optic Disc:
    • The point where fibers from the retina converge to form the optic nerve.
    • Located on the nasal side of the retina.
    • Has distinctive features related to color, shape, and margins, which are assessed clinically.
  • Retinal Vessels:
    • Each quadrant of the retina contains a paired artery and vein.
    • These vessels are directly observable in the living body, making the retina a unique location for assessing vascular health.
  • Macula:
    • Found on the temporal side of the fundus.
    • Appears slightly darker due to pigmentation.
    • Surrounds the fovea centralis, which is the site of the sharpest and most detailed vision.
    • Responsible for receiving and processing light from the central visual field.

👉 Key Points

  • The retina transforms light into neural signals, allowing vision.
  • Key visible structures: optic disc, retinal vessels, background, and macula.
  • The optic disc is the origin of the optic nerve, located nasally.
  • Retinal vessels consist of paired arteries and veins extending into each quadrant.
  • The macula, with its central fovea, provides the sharpest visual acuity.

📝 Detail Information

The retina contains specialized photoreceptor cells: rods (for night vision and peripheral vision) and cones (for color and fine detail). The optic disc creates a natural “blind spot” because it lacks photoreceptors. Clinical assessment of the optic disc can reveal signs of increased intracranial pressure (papilledema) or glaucoma (increased cup-to-disc ratio). The retinal vessels can show changes associated with systemic hypertension or diabetes. The macula, though small, is essential for tasks requiring fine visual discrimination such as reading or recognizing faces. Degeneration of the macula, as seen in age-related macular degeneration (AMD), leads to loss of central vision while peripheral vision remains intact.

Visual Pathways and Visual Fields

Light entering the eye is refracted by the transparent structures of the cornea, aqueous humor, lens, and vitreous body before striking the retina. The retina then transforms the light stimulus into nerve impulses, which are transmitted to the brain’s visual cortex for interpretation.

  • Image formation: The image projected onto the retina is upside down and reversed relative to the object being viewed.
  • Optic nerve formation: All retinal fibers converge to form the optic nerve while maintaining their spatial arrangement.
  • Optic chiasm: At this crossing point, fibers from both eyes intersect. Fibers from the nasal half of each retina cross to the opposite side, while fibers from the temporal half remain on the same side.
  • Visual field processing:
    • The left optic tract carries fibers from the left half of each retina.
    • The right optic tract carries fibers from the right half of each retina.
    • As a result, the right side of the brain processes the left visual field, and the left side of the brain processes the right visual field.

Visual Reflexes

The eye maintains function and clarity through several automatic reflexes:

  • Pupillary Light Reflex (Pupil reacts to light)
    • Normal constriction of the pupils in response to bright light striking the retina.
    • Controlled by a subcortical reflex arc, meaning it occurs without conscious control.
  • Fixation (You fix your attention on something)
    • A reflex that directs the eyes toward an object attracting attention.
    • Ensures the image is centered on the fovea centralis, where vision is sharpest.
  • Accommodation
    • Adaptation of the eye for near vision.
    • While the lens itself cannot be directly observed, two outward signs of accommodation can be seen:
      • Convergence: Inward turning of both eyes toward the near object.
      • Pupillary constriction: Reduction in pupil size to improve focus.

👉 Key Points

  • Light is refracted through the cornea, aqueous humor, lens, and vitreous before reaching the retina.
  • Images are inverted and reversed on the retina.
  • At the optic chiasm, nasal fibers cross, allowing each side of the brain to process the opposite visual field.
  • Major reflexes include:
    • Pupillary light reflex (automatic pupil constriction).
    • Fixation (directing gaze to an object of interest).
    • Accommodation (adjustment for near vision with convergence and pupillary constriction).

📝 Detail Information

The crossing of fibers at the optic chiasm is critical for binocular vision. Lesions at different points along the visual pathway produce characteristic patterns of visual field loss (e.g., optic nerve lesion causes monocular blindness, chiasm lesion causes bitemporal hemianopia). The pupillary light reflex is used clinically to assess midbrain function; its absence can indicate severe neurological damage. Accommodation deteriorates with age due to loss of lens elasticity, leading to presbyopia. Fixation may be disrupted in conditions such as nystagmus, where the eyes exhibit involuntary, repetitive movements.

Developmental Competence: Aging Adults

As people age, several changes in ocular anatomy and function occur:

  • Pupil size gradually decreases. This reduces the amount of light entering the eye, making older adults more sensitive to low-light conditions.
  • Presbyopia: The lens loses elasticity, becoming hard and glasslike. This loss of flexibility decreases the ability of the lens to change shape for accommodation, resulting in difficulty focusing on near objects.
  • By approximately age 70, the normally transparent lens fibers begin to thicken and yellow. This marks the early development of cataracts.
  • Visual acuity often declines gradually beginning after age 50, with more pronounced deterioration after age 70.
Condition
Clinical Effect
Pathophysiology
Cataract Formation
Cloudy vision, glare sensitivity
Clumping of lens proteins causing lens opacity
Glaucoma
Peripheral vision loss, eventual blindness if untreated
Increased intraocular pressure damages optic nerve; most often chronic open-angle glaucoma
Age-related Macular Degeneration (AMD)
Loss of central vision, difficulty with reading/recognition
Breakdown of macular cells
Diabetic Retinopathy
Leading cause of blindness in adults aged 25–74
Microvascular retinal damage due to diabetes

Culture and Genetics

  • Iris and retinal pigmentation vary across populations, affecting both appearance and possibly disease risk.
  • Cataracts: Leading cause of global blindness. Roughly 80% of cases are preventable or curable with surgery.
  • Glaucoma: Prevalence increases with age.
    • African Americans are 3–6 times more likely to develop glaucoma compared to Caucasian Americans.
    • Primary angle glaucoma is a leading cause of blindness in African Americans and Hispanics.
    • A family history of glaucoma significantly increases risk.
  • Age-related macular degeneration (AMD):
    • Most common in U.S. adults over 75.
    • More prevalent in Caucasians.
    • Risk factors include family history, cigarette smoking, hyperopia (farsightedness), light iris color, hypertension, high cholesterol, and female gender.
  • Visual Impairment (VI):
    • Defined as inability to see letters at 20/50 or below on the eye chart.
    • Expected to double in prevalence in the U.S. due to the aging population.
    • Most commonly affects Caucasian women and older adults, but rates are increasing among African Americans and Hispanics.
    • Routine visual screening is crucial for safety and early detection.

👉 Key Points

  • Aging leads to smaller pupils, presbyopia, cataracts, and gradual decline in visual acuity.
  • The top four causes of vision loss in older adults are cataracts, glaucoma, AMD, and diabetic retinopathy.
  • Cataracts are largely treatable; glaucoma and AMD require lifelong management.
  • African Americans and Hispanics have higher risk for glaucoma; Caucasians have higher rates of AMD.
  • Visual impairment is expected to rise, emphasizing the need for regular screening.

📝 Detail Information

Presbyopia typically begins in the 40s and is managed with corrective lenses (reading glasses or bifocals). Cataract surgery is one of the most common and successful surgical procedures worldwide. Glaucoma screening is particularly important for African Americans, given earlier onset and higher risk. AMD is often categorized as "dry" (atrophic, more common) or "wet" (neovascular, more severe). Preventive measures such as smoking cessation, controlling blood pressure, and maintaining healthy cholesterol levels may reduce risk. Diabetic retinopathy progression can be slowed through strict blood glucose control and regular eye exams.

Subjective Data: Eye Assessment

When collecting subjective data, the nurse must ask focused questions to assess the patient’s visual function, history, and risk factors. These questions reveal potential pathologies and guide the physical examination.

Vision Difficulty

Patients may report:

  • Decreased acuity (blurred vision or difficulty focusing).
  • Blind spots (scotomas).
  • Blurring of objects or clouding.
  • Halos, rainbows, or rings around lights, which may indicate corneal edema or glaucoma.
  • Loss of peripheral vision, commonly seen in glaucoma.
  • Night blindness, which can result from retinal degeneration, vitamin A deficiency, or glaucoma.

Assessment Questions:

  • Do you have difficulty seeing or any blurring?
  • Did it come on suddenly or gradually? One eye or both?
  • Is it constant, or does it come and go?
  • Do objects appear out of focus?
  • Do you notice moving spots, halos, or blind spots?

Pain

Pain may indicate infection, trauma, or intraocular pressure changes.

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Assessment Questions:
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Strabismus, Diplopia, Redness, and Swelling

Assessment Questions:

  • Do you have a history of crossed eyes? Now or in the past?
  • Do you ever see double? If so, is it constant or intermittent? One eye or both?
  • Do you experience redness or swelling?
  • Do these symptoms occur seasonally, or at particular times of the year?

Discharge and Past History

  • Discharge may be watery (tearing) or mucopurulent (infection).
  • Morning crusting can make opening the eyes difficult.
  • Past history of eye injury, surgery, or allergies increases risk for chronic ocular issues.

Assessment Questions:

  • Do you experience watering or excessive tearing?
  • Is there discharge? If so, what color?
  • How do you remove eye matter?
  • Have you had any injury, surgery, or allergies involving the eyes?

Glaucoma, Glasses, and Contact Lenses

  • Glaucoma: A major cause of blindness; early detection is critical.
  • Eyewear: Determines effectiveness of correction and possible complications.

Assessment Questions:

  • Have you ever been tested for glaucoma? What were the results?
  • Do you have a family history of glaucoma?
  • Do you wear glasses or contact lenses? How do they work for you?
  • When was your last prescription checked? Was it changed?
  • If you wear contact lenses:
    • Do you experience pain, photophobia, watering, or swelling?
    • How do you care for your lenses (cleaning, replacement, removal habits)?

Patient-Centered Care

This section emphasizes lifestyle and adaptation.

Assessment Questions:

  • When was your last vision test? Have you ever been tested for color vision?
  • Are there environmental hazards at home or work that affect your eyes? Do you use goggles or protection?
  • What medications do you take (systemic, topical, or eye-specific)?
  • Do you smoke?
  • If you have vision loss, how do you cope (large print, audiobooks, braille)?
  • Do you maintain a stable living environment to adapt to vision loss?
  • Do you fear complete blindness?

👉 Key Points

  • Subjective eye assessment includes vision difficulty, pain, strabismus/diplopia, redness/swelling, discharge, history, glaucoma risk, corrective lenses, and coping strategies.
  • Focused questions help distinguish acute conditions (infection, trauma) from chronic disease (glaucoma, AMD, diabetic retinopathy).
  • Patient-centered care addresses safety, adaptation, and psychological well-being.

📝 Detail Information

Subjective assessment often provides the first clues to ocular disease. For example, sudden vision loss with pain can suggest acute angle-closure glaucoma or retinal detachment, while gradual, painless loss is typical of cataracts or open-angle glaucoma. Reports of halos point to corneal edema, and diplopia may indicate cranial nerve palsy. Asking about coping strategies is crucial because vision loss significantly impacts independence, mental health, and quality of life. Preventive counseling—such as smoking cessation, workplace eye protection, and regular eye exams—is essential in long-term eye health.

Additional History for Aging Adults

When assessing older adults, targeted history questions help reveal functional and pathological vision changes:

  • Functional difficulties: Ask if the patient has trouble with tasks such as climbing stairs or driving, which may indicate depth perception or peripheral vision loss. Night vision is often impaired with aging due to smaller pupils and lens changes.
  • Glaucoma screening: Ask when they were last tested for glaucoma. Inquire about aching pain around the eyes, loss of peripheral vision, and how they manage eyedrops if diagnosed.
  • Cataract history: Ask if there is any history of cataracts or progressive blurring of vision.
  • Ocular comfort: Ask if the eyes feel dry or burning, and what remedies the patient uses (e.g., artificial tears, humidifier use).
  • Impact on activities: Assess if visual changes have reduced participation in daily activities such as reading, sewing, or driving, as these may reflect declining acuity or contrast sensitivity.

Objective Data

Preparation for Examination

  • Positioning: Begin vision screening with the patient standing. For detailed assessment, have the patient sit upright with their head at examiner’s eye level.
  • Equipment:
    • Snellen eye chart (for distance acuity).
    • Handheld vision screener (e.g., Jaeger card for near vision).
    • Opaque card or occluder (to test one eye at a time).
    • Penlight (for pupillary reflexes and corneal light reflex).
    • Applicator stick (for lid eversion if needed).
    • Ophthalmoscope (for fundoscopic examination).

Test of Central Visual Acuity

The Snellen chart is the most common and accurate method for testing distance vision.

Procedure:

  1. Place the chart in a well-lit location at eye level.
  2. Position the patient exactly 20 feet from the chart.
  3. Provide an opaque card to cover one eye at a time.
  4. Instruct the patient to read the smallest line of letters possible, encouraging an attempt at the next smaller line.
  5. If the patient wears glasses or contact lenses, they should keep them on (except for reading glasses).

Recording results:

  • If unable to read the largest letters at 20 feet, shorten the distance until the patient can see, and record the new distance (e.g., 10/20).
  • If acuity is too poor for letters, test if the patient can count fingers at a given distance or detect light perception with a penlight.

Test of Near Vision

Used for patients reporting difficulty with reading or close tasks.

Procedure:

  1. Use a Jaeger card (handheld screener with various print sizes).
  2. Hold the card about 35 cm (14 inches) from the eye, approximating the reading distance at which print equals that of a 20-foot chart.
  3. Test each eye separately, with corrective glasses on if normally used.
  4. A normal result is 14/14 in each eye, read clearly without hesitation or moving the card.
  5. If a screening card is unavailable, a magazine or newspaper can substitute.

Confrontation Test (Peripheral Vision)

This test compares the patient’s peripheral vision to the examiner’s.

Procedure:

  1. Sit facing the patient at eye level, about 2 feet apart.
  2. Have the patient cover one eye with an opaque card while you cover your opposite eye.
  3. Direct the patient to look straight at your eyes.
  4. Hold a finger or pencil midway between you and the patient, and slowly move it inward from the periphery in several directions.
  5. Ask the patient to say “now” as soon as they see the target.

Normal results:

  • 50° upward
  • 90° temporal
  • 70° downward
  • 60° nasal

Sensitivity can be improved by using a wiggling finger or a moving red object as the target.

👉 Key Points

  • Aging adults should be asked about functional problems (stairs, driving, night vision), glaucoma history, cataracts, ocular comfort, and daily activity impact.
  • Essential equipment for an eye exam includes the Snellen chart, Jaeger card, occluder, penlight, and ophthalmoscope.
  • Snellen chart provides central visual acuity; record findings in 20/x format.
  • Jaeger card tests near vision; normal is 14/14.
  • Confrontation test evaluates peripheral vision; normal field limits are 50° up, 90° temporal, 70° down, 60° nasal.

📝 Detail Information

The Snellen chart is a standardized tool, with 20/20 vision indicating normal acuity at 20 feet. Values like 20/40 mean the patient sees at 20 feet what a normal eye sees at 40 feet. Presbyopia, common after age 40, often first presents as difficulty with Jaeger card testing. The confrontation test is a gross screening method; formal perimetry provides a more accurate assessment of visual fields. Loss of temporal fields may suggest glaucoma, while bitemporal hemianopia often indicates a lesion at the optic chiasm.